AMAÇ: Bu çalýşmada laparoskopik kolesisrektomik uygulanan hastalarda kardiovasküler değişiklikler ve kan gazlarýnýn incelenmesi amaçlandý.
MATERYAL VE METOD: Tiyopenron, atrakuryum, isofluran, N2O + 02 ile genel anestezi verilmiş ASA 1 ve 2 grubuna dahil 24 laparoskopik kolesistektomi olgusu üzerine çalý­şýldý. Radial arter katerizasyonu uygulandý. Preoperatuvar, indüksiyon sonrasý, intraperitoneal CO2 insflasyonu sonrasý insuflasyondan 60 dakika sonra ve ekstübasyonda olmak üzere 5 kez, kalp tepe atýmý (KTA). ortalama arter basýncý. (OAB) kaydedildi ve arter kan gazlarý analizleri (AKGA) yapýldý. AKGA ile arteryel pH. PaO2, PaCO2, HCO3 ve BE saptandý.
BULGULAR: İnsuflasyon sonrasý bütün ölçümlerde KTA,
OAB ve PaCO2de anlamlý bir artýş(p<0.05), pH'da belirgin bir düşüş görüldü (p<0.00/ ). Insuflasyondan 60 dakika sonra HCO3 deðerleri anlamlý bir azalma gösterirken, BE deðerleri negatif yönde anlamlý yüksek bulundu (p<0.05).
SONUÇ: LK anestezisinde hasta seçiminde dikkatli olunmasý ne ortaya çýkabilecek komplikosyonlarý önlemek için yakýn bir kardiovasküler sistem ne kan gazý takibinin önemi vurgulandý.

OBJECTIVE: In this study cardiovascular and arterial blood gas changes during laparoscopic cholescystectomy were investigated.
STUDY DESIGN: This study was based on 24 ·patients­ who underwent lapuroscopic cholecysrectomy (LC). Thiopentone, atracurium, isoflurone, N2O + O2] were used for general anesthesia. In all patients radial arter was catheterisated. Heart rate (HR), mean arterial blood pressure (MABP) and arterial blood gazes were measured in preoperative period, after indication just after jýeritýmeal insufflation of CO2 60 minutes after insufflation and at extubation. Arterial blood gazes analyses covered arterial pH PaO2, PaCO2, HCO3 and BE.
RESULTS: After insufflation, in all measurements HR, MABP PaCO2 were increased (p<0.05). and pH decreased significantly (p<0.001). HCO3 value 60 minures after insufflation was decrased, BE value was increased in negative way (p<0.05).
CONCLUSION: We conclude that the complications of LC can he avoided by a careful monitoring of cardiovascular system and arterial blood gazes.

AMAÇ: Bu çalýşma esansiyel hipertansiyonlu ve aterosklerozu olan normotansif hasralarda endotel disfonksiyonunun bir göstergesi olan plazma endotelin-1 (ET-1) düzeylerinin saptanmasý ve saðlýklý kişilerin düzeyleri ile karşýlaştýrýlmasý amacý ile yapýlmýştýr.
MATERYAL VE METOD: 45 esansiyel hipertansiyonlu ve 15 normotansif atemskleroýik kormýer kalp hastasý ve 15 saðlýklý korýtml gruhýýnuýý plazma ET-1 düzeyleri radio immünassay (RİA) metodu ile saptandý.
BULGULAR: Esansiyel hipertansiyonlu hastalarda, hipetansiyonun şiddeti ve organ hasarý arttýkça ET-1 düzeylerininde anlamlý bir şekilde arttýğý saptandý. ET-1 düzeyleri ile yaş, hipertansiyonun süresi, vücut kitle indeksi, sistolojik ve diastolik kan basýnçlarý, sigara kullanýmý, plazma glikoz, total kolesterol, LDL kolesteml düzeyleri ile pozitif korelasyon, HDL kolesterol düzeyleri ile negatif korelasyon saptandý. Aterosklerotik koroner kalp hastalýğý olan normotansif hastalarda da plazma ET-1 düzeyleri anlamlý derecede yüksek bulundu.
SONUÇ: Endotel disfonksiyonunun bir göstergesi olan ET-1, her iki hastalýðýn etyopatogenezinde ve komplikasyonlarýn gelişiminde önemli rol oynamaktadýr.

OBJECTIVE: The aim of this study was to determine the levels of endothelin-1 (ET-1) which is indicator for disfunction of endoethelin at the normotensive patients who have essential hypertension and atherosclerosis and to compare with the levels of ET-1 in healty people.

STUDY DESIGN: The levels of plasma ET-1 is determined at the 45 patients who have a essential hypertansion and 15 patients who have normotensive atherosclerotic coronary heart disease and 15 healthy people with the met­hod of radio immunassay (RİA).

RESULTS: It is determined that when the intensity of hypertension and damage of the organ are increased at the patients who have essential hypertension, the fevels of ET-1 increased significantly too. There was a positive correlation between the levels of ET-1 and age,duration of hyper­tension, body mass index, sistolic and diastolic blood pressure, smoke, the levels of plasma glucose, total cholestem/ and negative correlation with the levels of HDL choleste­rol. The levels of plasma ET-1 was high significantly at the normotensive patients who have atherosclerotic coronary heart disease.

CONCLUSION: ET-1 wlýich is an indicator of disfunction of endothel plays an important role in the etiopatogenes of both disease and the development of complications.

AMAÇ: lg A nefropatisi en sýk görülen primer glomerulonefrittir. Bununla beraher tedavi yaklaşýmý konusunda henüz kesin bir görüş birliği sağlanamamýştýr.Bu çalýşmada lg A nefropatisinde immunsupressif tedavinin etkinliğini araştýrdýk.
MATERYAL VE METOD: Böbrek biopsisi ile tanýsý konmuş yaşlarý 15-62 arasýnda değişen, 9'u erkek, 5'i kadýn toplam 14 hasta deðerlendirildi. Böbrek fonksiyonlarý normal bulunan 12 hastaya (Cer 74-120 mi/dk) dört ay 'süre ile "prednisolorýe" (40 mg/gün) ve "azathioprine" (100 mg/gün) uygulandý. Başlangýçta renal yetersizliği olan (Ccr<60 ml/dk) iki hastaya "prednisolone" (40 mg/gün) ve "cydophosphamide" ( I00 mg/gün) dört ay süresince verildi.
BULGULAR: Dört aylýk tedavi sorumda bir hasta dýşýnda tüm hastalarda hematüri kayboldu. Başlangýçta saptanan proteinüride (ortalama 1.25±0.39 gr/gün) tedaviden sonra (ortalama 0.11±0.08 gr/gün) anlamlý bir azalma görüldü (p<0.05 ). Proteinürisi olan dokuz hastanýn yedisinde proteinüride tam remisyon elde edildi; iki hastada proteinüride azalma saptanmadý. Başlangýçta renal yetersizliği olan iki hastada, tedaviden sonra böbrek fonksiyonlarýnda anlamlý bir düzelme saptandý (p<0.05). 1-6 yýl süre ile takip edilen hastalarýn birbirinde böbrek fonksiyonlarýnda azalma gözlenmedi.
SONUÇ: Uyguladýðýmýz immunsupressif tedavi ile elde ettiğimiz sonuçlar, lg A nefropatisinde immunsupressif tedavinin önemli bir yeri olduğu kanaatini uyandýrmýştýr.

OBJECTIVE: lg A nephropathy is the most common primary glomerulorýephritis, no consensus has emerged to permit a unified disease, spesific approach to treatment. In this study we evaluated the efficacy of immunosuppressive therapy in lg A nephropathy.
STUDY DESIGN: Our study was carried out in 14 patients with biopsy-proven lg A nepropatlýy (9 males and 5 females, aged between 15-62 years, mean aýe 30.5±14.1 years). Their presenting symptoms were macroscopic/microscopic hematuria aýýd proteinuria ranging between 0.5-2 gr/day. Twelve patients with normal renal function (Cer 74-120 ml/min) underwent combined therapy using prednisolone (40 mg/day) and azathioprine (100 mg/day) for four months. Two patients who displayed renal failure (Ccr<60 ml!mirý) were treated with prednisolone (40 mg/day) and cyclophosphamide (100 mg/day).
RESULTS: By four months of therapy hematuria disappeared in all patients except one. Seven of the nine patients with proteirýuria underwent total remission of proteinuria within four months. Two patients had no significant decrease in proteirýuria. Two patients with renal failure showed significant improvement in renal functiorn.
CONCLUSION: These data may suggest that immunosupressive therapy reveals encouraging results in the management of Ig A nephropatlýy.

AMAÇ: Bu çalýşma, yenidoğanýn değerlendirilmesinde uzun yýllardýr rutin olarak kullanýlan Apgar skorlamasý ile umbilikal kord kan gazlarý arasýndaki ilişkiyi saptamak ve yenidoðanlarda fizyolojik hipoksiyi ortaya koymak amacýyla planlanmýştýr.
MATERYAL VE METOD: Şişli Etfal Hastanesi Kadýn Doðum Kliniklerinde doğan 18 sağlýklý term bebeğin Apgar skorlamasý ile beraber umbilikal kord kan gazlarý tayinleri yapýldý.
BULGULAR: 1. ve 5. dakika Apgar skorlarý 7'nin üzerinde tespit edilen tüm bebeklerde umbilikal kord kaný pH değerleri ortalama 7.26 (7.19-7.39), P02 ortalama 23.36 (17.4-3 I.2) mnýHg, PC0 2 ortalama 45.49 (31.8-61.2), Base excess ortalama -6.00 (-1.00--/4.9) olarak saptandý.
SONUÇ: Literatür bilgilerine uygun olarak bulunan umbilikal kord kan gazlan değerleri sağlýklý yenidoğanlarda fizyolojik hipoksiyi tanýmlamada yararlý ve geçerli bir yöntemdir.

OBJECTIVE: This study is planned to confirm the relation between Apgar scores and umbilical cord blood gases and to assignee the physiological hypoxia of' newborn infants.
STUDY DESIGN: The Apgar scores and umbilical cord blood gases of 18 healthy term newborn infants, who were born at the Obstetric and Gynecology Department at Istanbul Şişli Etfal Hospital were selected.
RESULTS: Those infants who had Apgar scores over 7 at 1 and 5 minutes, had the average umbilical cord blood pH value 7.26 (7./9-7.39), the average P02 value 23.36 ( 17.4- 31.2) mmHg, the average PC02 value 45.49 (31.8-61.2) mmHg and the average hase excess value of-6.00 (-1.00-- 14.9).
CONCLUSION: These results, which are the same as the scentific knowledge, proves us that the umbilical cord blood gases is an effective and current method, to describe the newborn physiological hypoxia

AMAÇ: Bu çalýşma bozulmuş glikoz toleransý (BGT) ve ateroskleroz arasýndaki ilişkiyi araştýrmak,akut myokard enfarktüsü sonrasý meydana gelen glikoz metabolizma deðişikliklerini izlemek, akut myokard enfarktüsü geçiren hastalarda, metabolik sendromun diğer parametreleri ile bozulmuş glikoz toleransý arasýndaki ilişkile­ri deðerlendirmek üzere yapýldý.
MATERYAL VE METOD: Şişli Etfal Hastanesi 2. Dahiliye Servisi ve Koroner Yoðun Bakým Ünitesinde yatan hastalardan basit rastlantýsal örnekleme metodu ile seçilen ilk gruptaki 20 hastaya akut myokard enfarktüsü sonrasý 14-21. günler arasýnda, ikinci gruptaki 24 hastaya, akut myokard enfarktüsü sonrasý 16-18 ay sonrasý oral glikoz tolerans testi (OGTT) yapýldý. Kontrol grubu olarak 24 sağlýklý kişiye OGTT yapýldý. Ayrýca ilk gruptaki hastalardan BGT saptanan 7 tanesine 3 ay sonra OGTT tekrar yapýldý.
BULGULAR: İlk gruptaki 20 hastanýn 13 tanesinde (%65) BGT saptanýrken, ikinci gruptaki 24 hastanýn 13 tanesinde (%54) BGT saptandý. Kontrol gruhundaki 16 hastanýn 1 tanesinde BGT saptandý. Kontrol grubu ile her 2 çalýşma gruhu arasýnda istatistiksel olarak ileri derecede anlamlý fark saptanýrken (p<0.001 p<l).001 ), çalýşma grýýptan arasýnda fark saplanmadý (p>0.5). İlk gruptaki BGT saptanan 13 hastadan, OGTT tekrarlanabilen 7 hastadan, 6 tanesinde BGT’nin devam ettiðini, 1 hastada ise diabetes mellitus geliştiði saptandý. Çalýşma gruplarý birleştirilerek, BGT ile metabolik sendromun diðer parametreleri olan android ·ohl'Zite, hipertansiyon, total kolesterol, trigliserid, LDL ve VLDL kolesterol düzeyleri ile pozitif korelasyon, HDL kolesterol düzeyleri ile negatif korelasyon saptandý.
SONUÇLAR: 811 bulgulara göre; aterosklerotik koroner kalp hastalýðý olanlarda BGT bulunma oranýnýn normal kişilere göre ileri derecede yüksek olduðunu, BGT’nýn ateroskleroz için bir risk faktörü olarak kabul edilmesi gerektiðini söyleyebiliriz. Ayrýca akut myokard enfarktüsü sonrasý, 2 hafta sonrasýnda glikoz metabolizmasýnda deðişiklik olmadýðýný, BGT’nýn metabolik sendromun diðer parametreleri ile bir arada bulunduðunu, aterosklerotik koroner kalp hastalýðý olan hastalarda risk faktörleri araştýrýlýrken BGT'nin de araştýrýlmasý gerektiðini savunuyoruz.



OBJECTIVE: This study is made to investigate the relationship between the impaired glucose tolerance (IGT) and atherosclerosis, to see the metabolic changes of glucose after myocardial infarction and to take into consideration the relationship between the IGT and other parameters of metabolic syndrom in patients with post myocardial infarction.
STUDY DESIGN: 2 groups of patients are selected from Şişli Etfal Hospital, II. Medicine Clinic and Coronary Intensive.Care Unit with simple sampling method. For the with acute myocardial infarction in the first group oral glucose tolerance test (OGTT) was made between the 14-21 days and for the other 24 patients­ with acute myocardial in farction in the second group. OGTT was made 16-18 month later. As for the control, group 24 healthy sample was selected and OGTT was made. Mean while for the 7 patients in the first group with the IGT.·OGTT was repeated 3 months later.
RESULTS: In the first group 13 patients out of 20 (65%)had IGT, in the second group 13 patients out of 24 (54%) had IGT. in the control group I patient out of I6 had IGT. Between the control group and the other two groups, there has been a great difference statistically (p<0.001, p<0.001). On the other hand no difference between the study groups has been seen(p>0.5 ). Gathering the results of study groups a positive correlation has been detected between the IGT and other parameters of metabolic syndrom like android obesity, hypertension, blood levels of total cholestaol, trigliserid, LDL and VLDL cholesterol. As well as that a negative correlation has been detected between the IGT and blood levels of HDL cholesterol.
CONCLUSION: According to these results we can say that patients with the atherosclerotic coronary heart disease have more frequent IGT with respect to normal people and IGT is a risk factor for atherosclerosis. Also we detected no change in glucose metabolism 2 weeks after myocardial infarction and we claim that IGT is among the parameters of metabolic syndrom and there is need to investigate IGT with the other risk factors in patients with the atherosclerotic coronary heart disease.

Çocuklarda premedikasyon amacý ile oral yolla Midazolam, Triclofos sodium, Alfentanyl kullanarak etkilerini plasebo ile karşýlaştýrdýk.
Çalýşmaya 1-10 yaş arasý ASA I/II grubundan 53 hasta dahil edildi.
Deðerlendirmede sodasyon skalasý, ebeveynlerinden ayrýlmadaki tavýrlarý, indüksiyon yöntemine gösterdikleri tepki (IV-Maske), Sistolik Arter Basýncý (SAB),Diastolik Arter Basýncý (DAB), Kalp Atým Hýzý, sP02 ve kusma parametreleri göz önüne alýndý.
Premedikosyon uygulanan 3 grup hastada sedasyon skalasý plaseboya göre ileri derecede anlamlýydý.
Midozolam ve Alfentanyl kullanýlan çocuklar ebeveynlerinden ayrýlmada plaseboya göre ileri derecede rahattýr.
Çonýklarda oral premedikasyonda yüksek sedasyon etkisi ve yan etkisinin azlýðý nedeniyle Midazolomu önermekteyiz.

ln 53 children patients aged between 1-10 years and ASA I-II, we compared the effects of peroral applicated Midazolam, Triclofos sodium Alfentanyl versus placebo, as premedicant agents.
For evaluation sedation scale, atitudes while separeted from parents, reactions to induction methods (IV or mask applications), Systolik and Diastolik Arterial Pressure (SAP and DAP), heart rate, sP02 and vomiting episodes were examined.
According to the placebo, sedation scales were significant evident in the 3 groups and children who applicated Midazolam or Alfentanyl were calm while separated from parents.
We can propose Midazolam causing less side effects and more sedation as orally premedicant agent in children.

AMAÇ: Lireratürde ACE İnhibitörlerinin son yýllarda nefrotik sendromlu hastalarda proteinüri üzerine olumlu etkisi olduðuna dair yayýnlara rastlanmaktadýr. Kliniðimizde nefrotik sendrom vakalarýnda, renal amiloidoza oldukça sýk rastlanmaktadýr (32%). Bu nedenle diðer nefirotik sendromlar yanýnda ACE inhibitörlerinin, renal amiloidozda proteinüri üzerine etkisini araştýrmak istedik.
MATERYEL VE METOD: Çalýşmaya, tanýsý böbrek biyopsisi ile konmuş, ciddi proteinürisi olan (>3 gr/gün) 9 renal amiloidozlu hasta alýnmýştýr. Başlangýçta 1 hasta dýşýnda diðerlerinin böbrek fonksiyonlarý normaldi. Hastalarýn tümü normotansifti. Kaptopril (25-100 mg/gün) Tedavi verilen hastalar 1-19 ay (ortalama: 7.12 ay) takip edildi.
BULGULAR: Başlangýçta saptanan proteinüride (ortalama: 7.86±4.4 gr/gün) tedavi ile (ortalama: 6.16±4.12 gr/gün) saptanan azalma istatistiki açýdan anlamlý bulunmadý (p>0.05 ). Bir hasta ciddi hipertansiyon geliştiði için çalýşmadan çýkarýldý. Tedavi sýrasýnda 2 hastada ortostatik hipotansiyon gelişti. Serum üre, kreatinin, Na, K deðerlerinde tedavi ile anlamlý bir deðişiklik olmadý.
SONUÇ: Ciddi proteinürisi olan 8 renal amiloidozlu vakada kaptopril tedavisi ile proteinüride anlamlý bir azalma olmadý.

OBJECTIVE: Although ACE inhibitors have heen used to reduce proteinuria in patients with nephrotic syndrome in recent years, their effect in renal amyloidosis has not yet been investigated. Nephrotic: syndrome due to renal amyloidosis is very frequent (32%) in our statistics. Therefore we intended to study the effect of ACE inhbitors on proteinuria in patients with nephrotic syndrome due to renal amyloidosis.
STUDY DESIGN: Nine patients (aged between 18-49 years, 5 males and 4 females) were included to our study. All the patients were normotensive and had normal renal function except one. The patients were commenced on captopril (25-100 mg/day) and were followed up for 1- 19 months (mean follow-up period was 7 months).
RESULTS: At the end of the follow-up period peroteinuria decreased from 7.96±4.42 g/day to 6.16±4./2 g/day. The decrease in proteinuria was not statistically significant (p>0.05). No signifýcant changes in serum urea, creatinine, sodium aýýd potassium levels were observed. One patient was withdrawn from the study because of severe hypotension. In two patients orthostatic hypotension developed during the treatment.
CONCLUSJON: This study may suggest that ACE inhibitors had no sigrýifýcarýt ejfý: ct oýý rerýal amyloidosis witlý lýeavy prrýteiýýýýria. Orthostatic hypoteýýsimý appeared to he a major problem in the treatment with ACE inhibitors.

AMAÇ: Fetüs mesanesindeki idrar volümünün amniotik sývýya olarý katkýsýný araştýrdýk.
MATERYEL VE METOD: Polikliniğe başvuran 118 gebede yapýlmýştýr. Ultrasonografik olarak dört kadran amniotik sývý indeksi tespit edildi ve fetüs mesanesi en uzun çapýndan ölçüldü. İkinci ölçüm birinci ölçümü takiben 1 saat sonra yapýldý. Elde edilen sonuçlar studen-t testiyle istatistiksel olarak deðerlendirildi.
BULGULAR ve SONUÇ: Fetüs mesane volümünde istatistiksel olarak anlamlý derecede azalma saptanan olgularda antiotik sývýdaki artýşda anlamlý idi.

OBJECTIVE: We assess the effect of urinary volum in fetal bladder to amniotik fluid volume.
MATERIAL AND METHOD: Our study group consisted of 118 patients who applied to our polclinic. Amniotic fluid volume in four quadrants and tize greatest diameter of fetal urinary bladder were measured ultrasonographically. The second measurement was performed 1hour after the first. Statistical assesment of the results was performed using student-t test.
CONCLUSION: The cases where statistically meaningful decrease in fetal urinary volume present, the increase in amniotic fluid volume was also meaningful.

AMAÇ: Bu çalýşmada parsiyel epilepsi tanýsý olarý 63 hastada Magnetik Rezonans (MR) görüntülemenin etyolojiyi belirlemedeki yeri araştýrýlmýştýr.
MATERYEL VE METOD: Şişli Etfal Hastanesi Nöroloji Kliniði Polikliniðinde parsiyel epilepsi tanýsý ile izlenen 63 hastanýn MR bulgularý retrospektif olarak deðerlendirildi. Hastalardaki nöbet tipleri hem kendilerinden alýnan anamnez, hem de nöbeti yakýndan izlemiş olan kişilerden edinilen bilgiler ýşýðýnda belirlendi. Hastalarýn tümünde elektroansefalografi (EEG) yýlda en az iki kez yinelendi. MR incelemesinde T1, T2 ve proton yoðunluðunda aksiyal,koronal ve sagital kesitler alýndý.
BULGULAR: Hastalarýn 32'si kadýn, 31 tanesi erkek olarak belirlendi. Nöbet tipi 6 hastada basit parsiyel, 14 hastada kompleks parsiyel ve 43 hastada sekonder jeneralize olarak saptandý. Elektroamefalografik (EEG) incelemelerde 47 hastada patoloji bulundu. 16 hastada ise EEG nor­maldi. MR 36 hastada patoloji gösterdi. MR bulgularý 10 hastada arroji, 5 lýastada mu/tipi ak madde lezyonu, 4 has- tada yer kaplayýcý le: ymý, 4 hastada gelişimsel anomali, 3 hastada lýipoksik aýýsejcýlopati, 2 hastada mezial temporal skleroz, 2 hastada ventrikül asimetrisi, 2 hastada eski kalsifiye lezyon, 1 hastada hidrosefali, 1 hastada vasküler lezyon, 1 hastada nörofibromatozis ile uyumlu multipl nodüler ak madde lezyonu olarak sýnýflandýrýldý. Her iki oksipital bölgede az çok simetrik hiperintens iki lezyonu olan hastaya ise kesin taný konulamadý.
SONUÇ: Parsiyel epilepsi tanýsý alanlarda görüntülemede ilk seçilecek yöntem MR olmalýdýr. MR bulgularý, kraniyal bilgisayarlý tomografide (BT) gösterilemeyen migrasyon anmýýa/ileri, mesial temporal skleroz ve demyelinizasyonla giden proçeslerin tanýnmasýna olanak sağlar.

OBJECTIVE: In this study we have investigated the role of magnetic resonance imaging (MRI) in determining the etiology in 63 patients with partial epilepsy.
MATERIALS AND METJIODS: The MRI findings of 63 patients with partial epilepsy followed by Epilepsy Outpatient Department of Şişli Etfal Hospital Neurology Clinic were investigated retrospectively. The type of seizure was detemýined by the informatiorý taken from the patients and their relatives who had seen the seizure. The electroecephalogram was recorded twice yearly in each patient. All patients were screened in T1, T2 and proton density.
RESULTS: Thirty-two patients were female and 31 were male. The seizure type was simpily partial in 6 patients, complex partial in 14 patients and secondarily generalised in 43. EEG was pathologic in 47 patients and normal in 16. Thirty-six of 63 patients had abnormal MRI. Ten patients had atrophy, followed by 5 multiple, small lesion in white matter, 4 space occupying leisons. 4 developmental abnormalities, 3 hypoxic encephalopaty, 2 mesial temporal sclerosis, 2 venturicular asymmetry, 2 calcified lesion. 1 hydrocephaly, 1 vascular lesion, 1 multipl hamartomatous lesions in white matter in accordance with neurofibromatosis. One patient with hyperintense symmetrical lesions in occipital lobe could not he classified.
CONCLUSION: We claim that MRI is a very important tool revealing the etiology in patients with partial seziures MRI allows us to determine the migration ubnormalities and demyelinating lesions which could not be shown in CT.